Supplementary Materials

Supplementary Material for:

Durable sequence stability and bone marrow tropism in a macaque model of human pegivirus infection

Adam L. Bailey, Michael Lauck, Mariel Mohns, Eric J. Peterson, Kerry Beheler, Kevin G. Brunner, Kristin Crosno, Andres Mejia, James Mutschler, Matthew Gehrke, Justin Greene, Adam J. Ericsen, Andrea Weiler, Gabrielle Lehrer-Brey, Thomas C. Friedrich, Samuel D. Sibley, Esper G. Kallas, Saverio Capuano III, Jeffrey Rogers, Tony L. Goldberg, Heather A. Simmons, David H. O'Connor*

*Corresponding author. E-mail: doconnor{at}primate.wisc.edu

Published 16 September 2015, Sci. Transl. Med. 7, 305ra144 (2015)
DOI: 10.1126/scitranslmed.aab3467

This PDF file includes:

  • Fig. S1. Parameters of SIV-induced disease in study animals.
  • Fig. S2. Lack of SPgV-induced disease in study animals.
  • Fig. S3. SPgVs exhibit little sequence variability in natural hosts.
  • Fig. S4. The tissue tropism of SPgV may vary depending on the duration of infection.
  • Table S1. Sequencing of SPgVs from wild baboons.
  • Table S2. Demographic information for macaques used in this study.
  • Table S3. Sequencing of SPgVmyb in cy0571.
  • Table S4. Sequencing of macaques infected with SPgVkob.
  • Table S5. SPgVmyb consensus-level variants in cy0571 at 219 dpi not found in inocula.

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