Research ArticleNanomedicine

RNAi targeting multiple cell adhesion molecules reduces immune cell recruitment and vascular inflammation after myocardial infarction

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Science Translational Medicine  08 Jun 2016:
Vol. 8, Issue 342, pp. 342ra80
DOI: 10.1126/scitranslmed.aaf1435

Article Information

vol. 8 no. 342 342ra80

PubMed: 
Print ISSN: 
Online ISSN: 
History: 
  • Received for publication December 22, 2015
  • Accepted for publication May 17, 2016
  • .

Author Information

  1. Hendrik B. Sager1,*,,
  2. Partha Dutta1,,
  3. James E. Dahlman2,3,4,,
  4. Maarten Hulsmans1,
  5. Gabriel Courties1,
  6. Yuan Sun1,
  7. Timo Heidt1,
  8. Claudio Vinegoni1,
  9. Anna Borodovsky5,
  10. Kevin Fitzgerald5,
  11. Gregory R. Wojtkiewicz1,
  12. Yoshiko Iwamoto1,
  13. Benoit Tricot1,
  14. Omar F. Khan3,
  15. Kevin J. Kauffman3,6,
  16. Yiping Xing2,3,
  17. Taylor E. Shaw2,3,
  18. Peter Libby7,
  19. Robert Langer2,3,4,6,
  20. Ralph Weissleder1,8,
  21. Filip K. Swirski1,
  22. Daniel G. Anderson2,3,4,6, and
  23. Matthias Nahrendorf1,9,*,
  1. 1Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA 02114, USA.
  2. 2Harvard–Massachusetts Institute of Technology (MIT) Division of Health Sciences and Technology, Cambridge, MA 02139, USA.
  3. 3David H. Koch Institute for Integrative Cancer Research, MIT, Cambridge, MA 02139, USA.
  4. 4Institute for Medical Engineering and Science, MIT, Cambridge, MA 02139, USA.
  5. 5Alnylam Pharmaceuticals, Cambridge, MA 02142, USA.
  6. 6Department of Chemical Engineering, MIT, Cambridge, MA 02139, USA.
  7. 7Cardiovascular Division, Department of Medicine, Brigham and Women’s Hospital, Boston, MA 02115, USA.
  8. 8Department of Systems Biology, Harvard Medical School, Boston, MA 02115, USA.
  9. 9Cardiovascular Research Center, Massachusetts General Hospital and Harvard Medical School, Boston, MA 02114, USA.
  1. *Corresponding author. Email: mnahrendorf{at}mgh.harvard.edu (M.N.); hendrik.sager{at}tum.de (H.B.S.)
    • These authors contributed equally to this work.

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