Research ArticleInflammation

Recruitment of classical monocytes can be inhibited by disturbing heteromers of neutrophil HNP1 and platelet CCL5

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Science Translational Medicine  09 Dec 2015:
Vol. 7, Issue 317, pp. 317ra196
DOI: 10.1126/scitranslmed.aad5330

Article Information

vol. 7 no. 317 317ra196

PubMed: 
Print ISSN: 
Online ISSN: 
History: 
  • Received for publication September 26, 2015
  • Accepted for publication October 29, 2015
  • .

Author Information

  1. Jean-Eric Alard1,*,
  2. Almudena Ortega-Gomez1,*,
  3. Kanin Wichapong2,
  4. Dario Bongiovanni3,
  5. Michael Horckmans1,
  6. Remco T. A. Megens1,2,
  7. Giovanna Leoni1,
  8. Bartolo Ferraro1,4,
  9. Jan Rossaint5,
  10. Nicole Paulin1,
  11. Judy Ng3,
  12. Hans Ippel2,
  13. Dennis Suylen2,
  14. Rabea Hinkel1,3,6,
  15. Xavier Blanchet1,
  16. Fanny Gaillard7,
  17. Michele D’Amico4,
  18. Phillipp von Hundelshausen1,
  19. Alexander Zarbock5,
  20. Christoph Scheiermann8,
  21. Tilman M. Hackeng2,
  22. Sabine Steffens1,6,
  23. Christian Kupatt3,6,
  24. Gerry A. F. Nicolaes2,,
  25. Christian Weber1,2,6, and
  26. Oliver Soehnlein1,6,9,
  1. 1Institute for Cardiovascular Prevention, Ludwig Maximilians University Munich, 80336 Munich, Germany.
  2. 2Department of Biochemistry, Cardiovascular Research Institute Maastricht, University Maastricht, 6200 Maastricht, Netherlands.
  3. 3Medizinische Klinik I, Technische Universität München, 81675 Munich, Germany.
  4. 4Department of Experimental Medicine, University of Naples, 80138 Naples, Italy.
  5. 5Department of Anesthesiology, University of Münster, 48149 Münster, Germany.
  6. 6German Centre for Cardiovascular Research, partner site Munich Heart Alliance, 80336 Munich, Germany.
  7. 7Roscoff Biological Station, Pierre et Marie Curie University, 29682 Paris, France.
  8. 8Walter-Brendel-Center of Experimental Medicine, Ludwig Maximilians University Munich, 81377 Munich, Germany.
  9. 9Department of Pathology, Academic Medical Center, 1105 Amsterdam, Netherlands.
  1. Corresponding author. E-mail: oliver.soehnlein{at}gmail.com
  • * These authors contributed equally to this work.

  • These authors contributed equally to this work.

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