Research ArticleCancer

A therapeutically targetable mechanism of BCR-ABL–independent imatinib resistance in chronic myeloid leukemia

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Science Translational Medicine  03 Sep 2014:
Vol. 6, Issue 252, pp. 252ra121
DOI: 10.1126/scitranslmed.3009073

Article Information

vol. 6 no. 252 252ra121

PubMed: 
Print ISSN: 
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History: 
  • Received for publication March 19, 2014
  • Accepted for publication July 9, 2014

Author Information

  1. Leyuan Ma1,
  2. Yi Shan2,
  3. Robert Bai1,
  4. Liting Xue3,
  5. Christopher A. Eide4,
  6. Jianhong Ou3,
  7. Lihua J. Zhu3,5,
  8. Lloyd Hutchinson6,
  9. Jan Cerny2,
  10. Hanna Jean Khoury7,
  11. Zhi Sheng8,9,
  12. Brian J. Druker4,
  13. Shaoguang Li2 and
  14. Michael R. Green1,*
  1. 1Howard Hughes Medical Institute, Programs in Gene Function and Expression and Molecular Medicine, University of Massachusetts Medical School, Worcester, MA 01605, USA.
  2. 2Division of Hematology/Oncology, Department of Medicine, University of Massachusetts Medical School, Worcester, MA 01605, USA.
  3. 3Programs in Gene Function and Expression and Molecular Medicine, University of Massachusetts Medical School, Worcester, MA 01605, USA.
  4. 4Howard Hughes Medical Institute, Knight Cancer Institute, Division of Hematology and Medical Oncology, Oregon Health and Science University, Portland, OR 97239, USA.
  5. 5Program in Bioinformatics and Integrative Biology, University of Massachusetts Medical School, Worcester, MA 01605, USA.
  6. 6Department of Pathology, University of Massachusetts Medical School, Worcester, MA 01605, USA.
  7. 7Department of Hematology and Medical Oncology, Winship Cancer Institute of Emory University, Atlanta, GA 30332, USA.
  8. 8Virginia Tech Carilion Research Institute, Roanoke, VA 24016, USA.
  9. 9Department of Biomedical Sciences and Pathobiology, Virginia Tech, Blacksburg, VA 24061, USA.
  1. *Corresponding author. E-mail: michael.green{at}umassmed.edu

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