Research ArticleAcute Coronary Syndromes

Inhibition of the Cardiomyocyte-Specific Kinase TNNI3K Limits Oxidative Stress, Injury, and Adverse Remodeling in the Ischemic Heart

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Science Translational Medicine  16 Oct 2013:
Vol. 5, Issue 207, pp. 207ra141
DOI: 10.1126/scitranslmed.3006479

Article Information

vol. 5 no. 207 207ra141

PubMed: 
Print ISSN: 
Online ISSN: 
History: 
  • Received for publication May 1, 2013
  • Accepted for publication August 28, 2013
  • .

Author Information

  1. Ronald J. Vagnozzi1,2,
  2. Gregory J. Gatto Jr.3,
  3. Lara S. Kallander3,
  4. Nicholas E. Hoffman2,
  5. Karthik Mallilankaraman2,
  6. Victoria L. T. Ballard3,
  7. Brian G. Lawhorn3,
  8. Patrick Stoy3,
  9. Joanne Philp3,
  10. Alan P. Graves4,
  11. Yoshiro Naito5,
  12. John J. Lepore3,
  13. Erhe Gao2,
  14. Muniswamy Madesh2 and
  15. Thomas Force2,6,*
  1. 1Program in Cell and Developmental Biology, Thomas Jefferson University, Philadelphia, PA 19107, USA.
  2. 2Center for Translational Medicine, Temple University School of Medicine, Philadelphia, PA 19140, USA.
  3. 3Heart Failure Discovery Performance Unit, Metabolic Pathways and Cardiovascular Therapeutic Area Unit, GlaxoSmithKline, King of Prussia, PA 19406, USA.
  4. 4Platform Technology and Sciences, GlaxoSmithKline, King of Prussia, PA 19406, USA.
  5. 5Cardiovascular Division, Department of Internal Medicine, Hyogo College of Medicine, Nishinomiya 663-8131, Japan.
  6. 6Cardiology Division, Temple University School of Medicine, Philadelphia, PA 19140, USA.
  1. *Corresponding author. E-mail: thomas.force{at}temple.edu

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