Editors' ChoiceAlzheimer’s Disease

Blaming an unusual suspect for endosomal traffic jams in Alzheimer’s disease

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Science Translational Medicine  21 Aug 2019:
Vol. 11, Issue 506, eaaz0730
DOI: 10.1126/scitranslmed.aaz0730

Abstract

Accumulation of β–C-terminal fragments, not β-amyloid, drives early endosomal alterations in familial Alzheimer’s disease human neurons.

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