Research ArticlePain

Pharmacological reversal of a pain phenotype in iPSC-derived sensory neurons and patients with inherited erythromelalgia

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Science Translational Medicine  20 Apr 2016:
Vol. 8, Issue 335, pp. 335ra56
DOI: 10.1126/scitranslmed.aad7653

Article Information

vol. 8 no. 335 335ra56

PubMed: 
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History: 
  • Received for publication October 29, 2015
  • Accepted for publication April 1, 2016

Author Information

  1. Lishuang Cao1,*,
  2. Aoibhinn McDonnell1,*,
  3. Anja Nitzsche1,*,
  4. Aristos Alexandrou1,
  5. Pierre-Philippe Saintot1,
  6. Alexandre J.C. Loucif1,
  7. Adam R. Brown1,
  8. Gareth Young1,
  9. Malgorzata Mis2,
  10. Andrew Randall3,
  11. Stephen G. Waxman4,
  12. Philip Stanley1,
  13. Simon Kirby1,
  14. Sanela Tarabar5,
  15. Alex Gutteridge1,
  16. Richard Butt1,
  17. Ruth M. McKernan1,
  18. Paul Whiting1,
  19. Zahid Ali1,,
  20. James Bilsland1,, and
  21. Edward B. Stevens1,,
  1. 1Pfizer Neuroscience and Pain Research Unit, The Portway Building, Granta Park, Cambridge CB21 6GS, UK.
  2. 2University of Bristol, School of Physiology, Pharmacology, and Neuroscience, Bristol BS8 1TD, UK.
  3. 3Hatherly College of Life and Environmental Sciences, University of Exeter, Prince of Wales Road, Exeter EX4 4PS, UK.
  4. 4Yale Center for Neuroscience and Regeneration Research, Veterans Affairs Medical Center, 950 Campbell Avenue, Building 34,West Haven, CT 06516, USA.
  5. 5Pfizer, 1 Howe Street, New Haven, CT 06511, USA.
  1. Corresponding author. E-mail: j.bilsland{at}ucl.ac.uk (J.B.); edward.stevens{at}pfizer.com (E.B.S.)
    • * These authors contributed equally to this work.

    • These authors contributed equally to this work.

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