Research ArticleNEUROBIOLOGY

Histone deacetylase inhibition rescues structural and functional brain deficits in a mouse model of Kabuki syndrome

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Science Translational Medicine  01 Oct 2014:
Vol. 6, Issue 256, pp. 256ra135
DOI: 10.1126/scitranslmed.3009278

Article Information

vol. 6 no. 256 256ra135

PubMed: 
Print ISSN: 
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History: 
  • Received for publication April 10, 2014
  • Accepted for publication September 11, 2014

Author Information

  1. Hans T. Bjornsson1,2,*,,
  2. Joel S. Benjamin1,3,,
  3. Li Zhang1,
  4. Jacqueline Weissman1,4,
  5. Elizabeth E. Gerber1,
  6. Yi-Chun Chen1,
  7. Rebecca G. Vaurio4,
  8. Michelle C. Potter5,
  9. Kasper D. Hansen1,6 and
  10. Harry C. Dietz1,7,8
  1. 1McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
  2. 2Department of Pediatrics, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
  3. 3Predoctoral Training Program in Human Genetics, McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
  4. 4Kennedy Krieger Institute, Baltimore, MD 21205, USA.
  5. 5Brain Science Institute, Neurology Department, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
  6. 6Department of Biostatistics, Bloomberg School of Public Health, Baltimore, MD 21205, USA.
  7. 7Division of Pediatric Cardiology, Department of Pediatrics, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
  8. 8Howard Hughes Medical Institute, Baltimore, MD 21205, USA.
  1. *Corresponding author. E-mail: hbjorns1{at}jhmi.edu
    • These authors contributed equally to this work.

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