Research ArticleHeart Failure

Histone deacetylase activity governs diastolic dysfunction through a nongenomic mechanism

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Science Translational Medicine  07 Feb 2018:
Vol. 10, Issue 427, eaao0144
DOI: 10.1126/scitranslmed.aao0144

Article Information

vol. 10 no. 427

PubMed: 
Print ISSN: 
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History: 
  • Received for publication June 5, 2017
  • Resubmitted September 7, 2017
  • Accepted for publication January 5, 2018

Author Information

  1. Mark Y. Jeong1,2,
  2. Ying H. Lin1,2,
  3. Sara A. Wennersten1,2,
  4. Kimberly M. Demos-Davies1,
  5. Maria A. Cavasin1,2,
  6. Jennifer H. Mahaffey1,2,
  7. Valmen Monzani3,
  8. Chandrasekhar Saripalli4,
  9. Paolo Mascagni3,
  10. T. Brett Reece5,
  11. Amrut V. Ambardekar1,2,
  12. Henk L. Granzier4,
  13. Charles A. Dinarello6 and
  14. Timothy A. McKinsey1,2,*
  1. 1Division of Cardiology, Department of Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
  2. 2Consortium for Fibrosis Research & Translation, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
  3. 3Italfarmaco S.p.A., Cinisello Balsamo, Italy.
  4. 4Department of Cellular and Molecular Medicine and Sarver Molecular Cardiovascular Research Program, University of Arizona, Tucson, AZ 85724, USA.
  5. 5Division of Cardiothoracic Surgery, Department of Surgery, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
  6. 6Division of Infectious Diseases, Department of Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
  1. *Corresponding author. Email: timothy.mckinsey{at}ucdenver.edu

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Aug 2018482428